Anti-Vascular Endothelial Growth Factor Receptor 1 (FLT1), Human Antibody

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产品名称: Anti-Vascular Endothelial Growth Factor Receptor 1 (FLT1), Human Antibody

产品型号: Y105090

产品展商: ABM

产品文档: 无相关文档

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Anti-Vascular Endothelial Growth Factor Receptor 1 (FLT1), Human Antibody

Anti-Vascular Endothelial Growth Factor Receptor 1 (FLT1), Human Antibody 的详细介绍

Overview	Print Version
Product Name	Anti-Vascular Endothelial Growth Factor Receptor 1 (FLT1), Human Antibody
Description	Rabbit polyclonal to VEGF Receptor 1. VEGF Receptor 1 (also known as FLT) belongs to the src gene family and shows tyrosine protein kinase activity that is important for the control of cell proliferation and differentiation. The protein acts as a receptor for VEGF, VEGFB and PGF. An alternatively spliced form of the gene produces a soluble protein (sFlt1) which binds vascular endothelial growth factor (VEGF) with high affinity. sFlt1 has a higher affinity for VEGF indicating that it may function as an inhibitor in the VEGF response. VEGF Receptor 1 is specifically expressed in most vascular endothelial cells and peripheral blood monocytes.
Subtype	polyclonal
Application	IHC
Raised In	Rabbit
Immunogen	Synthetic peptide - KLH conjugated
Guarantee	abm guarantees that all our Anti-Vascular Endothelial Growth Factor Receptor 1 (FLT1), Human Antibody will perform as described on this product webpage, if this is not the case we will provide you with a one-time replacement at no extra cost. Documentation and explanation of experiment conducted will be required when submitting a claim for replacement.
Properties
Formulation	Liquid
Quantity	50 μg
Clone	Polyclonal Antibody
Storage	This product is stable for several weeks at 4°C as an undiluted liquid. Dilute only prior to immediate use. For extended storage, aliquot contents and freeze at -20°C or below. Avoid cycles of freezing and thawing. Expiration date is one (1) year from date of receipt.
Purity	Affinity purified
Species Reactivity	Human
Specific References
Y105090 has not yet been referenced specifically in any publications.

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